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Conolidine’s analgesic results stem from its interaction with non-opioid pain pathways. Compared with opioids, which bind to µ-opioid receptors in the central nervous process, conolidine modulates alternate molecular targets. A Science Improvements research identified that conolidine interacts With all the atypical chemokine receptor ACKR3/CXCR7
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Elucidating the exact pharmacological system of action (MOA) of naturally occurring compounds can be tough. Whilst Tarselli et al. (60) created the initial de novo artificial pathway to conolidine and showcased that this naturally taking place compound proficiently suppresses responses to each chemically induced and inflammation-derived pain, the p
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